WHY WAS BANNING INCANDESCENTS UNWISE?

On the Tetragrammaton podcast, I did in May, I told Mr. Rick Rubin and Dr. Andrew Huberman that blue light controls sexual dimorphism in humans. Unfortunately, this hit the editing floor in the podcast.

Retinoic acid is Vitamin A. Humans require retinoic acid (RA) for many developmental processes. We can synthesize RA through the metabolic breakdown of retinol (see picture below). RA is needed for signaling and transcriptional regulation for ocular, cardiac, pancreatic, and testicular development processes. RA can influence transcription by binding with receptor proteins to help regulate the transcription of certain genes important for these developmental processes in both sexes.

It is being reported in other studies like the one cited below. The etiology is the liberation from melanopsin by blue light in our light bulbs and screens, and that freed Vitamin A causes changes in neurons in the hypothalamus to change the sexuality or alter their changes in puberty. Technology can cause this via the light on screens. The next few slides from my talks over the last 15 years show you the story.

I have written about the biophysical details on Patreon. This change in puberty also explains why young adults afflicted by these issues are at much higher risk for developing vasovagal symptoms or POTS. See the picture from the blog below. Puberty in humans is initiated by hormonal changes in the adolescent body that trigger physical and behavioral changes to reach adult maturation. As these changes occur, some adolescents experience concerning pubertal symptoms associated with autonomic nervous system dysfunction (ANS). This is more prominent in tech abusers or those who live under extreme indoor lights (LED).

Thiamine is Vitamin B1. B1 is a water-soluble, sulfur-containing vitamin. Thiamine is an essential nutrient that is a cofactor for several enzymes, mostly with mitochondrial localization. Some thiamine-dependent enzymes are involved in energy metabolism and biosynthesis of nucleic acids, whereas others are part of the antioxidant machinery. The brain, ovaries and testis are highly vulnerable to thiamine deficiency during puberty due to its heavy reliance on mitochondrial ATP production during the growth spurt. Thiamine storage is decreased by technology abuse and the use of coffee, tea, and alcohol.

Thiamine deficiency is more evident during rapid growth with an unmyelinated brain. This is why POTS is exploding in humans from puberty through young adulthood.

Thiamine deficiency contributes to several conditions spanning from mild neurological and psychiatric symptoms (confusion, reduced memory, and sleep disturbances) to severe encephalopathy, ataxia, POTS, dysautonomia, congestive heart failure, muscle atrophy, and even death.

Vasovagal syncope (VVS) and Postural Orthostatic Tachycardia Syndrome (POTS) are common disorders of the ANS associated with puberty that are related to orthostatic intolerance and share similar symptoms. Young females have decreased orthostatic tolerance and a higher incidence of VVS and POTS than young males. As puberty is linked to changes in specific sex and non-sex hormones, and hormonal therapy sometimes improves orthostatic symptoms in female VVS patients, pubertal hormones likely play a role in the increased susceptibility of young females to autonomic dysfunction.

These findings are linked to the light that the last two generations have been forced to live under by narratives around energy being pushed by government decree. It is also being sculpted by Obama's phase-out of incandescent lights in 2009 for LED replacement. Biden's, Obama's VP in 2009, enacted the total ban. This began this August.

This was being done to the best of energy savings. This false narrative is not based on decentralized biological risks to children. My website and blog aim to review the key hormonal changes associated with female puberty, their effects on the ANS, and their potential role in predisposing some adolescent females to cardiovascular autonomic dysfunctions such as VVS and POTS.

Melanin biology is strongly protective of autonomic dysfunction and changes in sexual dimorphic changes that are possible in children. Increases in pubertal hormones such as estrogen, thyroid, growth hormone, insulin, and insulin-like growth factor-1 promote vasodilatation and decrease blood volume. This can lead to relative dehydration signaling in the baroreceptors of the circulatory system. This may be exacerbated by higher levels of progesterone, which suppresses catecholamine secretion and sympathetic outflow.

People forget that melanin destruction alters the production of norepinephrine, epinephrine, and dopamine. All of these are key catecholamine chemicals in the ANS of humans. The picture above shows you these direct links. Abnormal heart rate increases in POTS patients may be exacerbated by pubertal increases in leptin, insulin, and thyroid hormones acting to increase sympathetic nervous system activity and/or catecholamine levels.

SUMMARY

Given the coincidental timing of female pubertal hormone surges and adolescent onset of VVS and POTS in young women, coupled with the known roles of these hormones in modulating cardiovascular homeostasis, it is likely that female pubertal hormones play a role in predisposing females to VVS and POTS during puberty. This is how decentralized medicine SHOULD be allowed to operate inside the USA. Further research into the government mandates of our light environment is necessary to confirm the effects of female pubertal hormones on autonomic function and their role in pubertal autonomic disorders such as VVS and POTS. This is needed to inform the treatment and management of these debilitating disorders.

CITES

https://studyfinds.org/blue-light-early-puberty/

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6633611/