【MedChem Spark】Polarized C-H as Nonconventional Hydrogen Bond Donor(Issue 3)

【MedChem Spark】Polarized C-H as Nonconventional Hydrogen Bond Donor(Issue 3)

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By Jin Li

Canonical hydrogen bond donors, N-H and O-H, play key roles in interactions between molecules and proteins and exist in substantial drug and clinical candidate molecules, especially in kinase inhibitors which have a N-H forming a hydrogen bond with hinge region. Issues associated with canonical hydrogen bond donors are their potentially negative influences on aqueous solubility, permeability, glucosidation metabolism, etc. In order to circumvent this, C-H in heteroaromatic rings containing nitrogen is often used as nonconventional hydrogen bond donors due to the increased acidity of the involved C-H hydrogen. For example, C-H in pyrazine was found to form nonconventional hydrogen bonds with protein backbone and –COOH residues (Figure 1). [1]

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Figure 1. C-H in pyrazine ring as nonconventional hydrogen bond donors (PDB code: 4NNI, 3U4W respectively)


In order to discover BBB-penetrant LRRK2 inhibitors, the replacement of indazole N1-H in compound 24 with imidazo[1,5-a]pyridine core C-H in compound 25 and “reverse indazole” C3-H in compound 26 was explored. [2] Despite the anticipated loss in potency, significantly reduced P-gp efflux was observed for compound 26, positively differentiating this core. Further optimization of compound 26 afforded highly potent, selective and BBB-penetrant LRRK2 inhibitors. A nonconventional hydrogen bond between C3-H in indazole with backbone C=O of LRRK2 Glu85 was demonstrated in an X-ray structure of one of LRRK2 inhibitors bound to LRRK2 (Figure 2).

Figure 2. C3-H in “reverse indazole” was used as a nonconventional hydrogen bond donor. (PDB code: 8E80)


Polarization of the aryl C-H to increase the hydrogen bonding potential was believed to be necessary to bring about sufficient TYK2 activity and two-point binding. With this strategy in mind, several [5,6]-fused heteroaromatic systems, including imidazopyridine scaffold in compound 27, [1,2,4] triazoles [1,5]pyridine scaffolds in compound 28, [1,2,4]triazole[4,3]pyridine scaffold in compound 29 and pyrazole[1,5]pyrazine scaffold in compound 30, were designed and evaluated (Figure 3). [3] Among the above scaffolds, the pyrazole[1,5]pyrazine scaffold was of particular interest due to the calculated increased polarization of the C-H for hydrogen bond donor properties. Consistent with the calculation result, pyrazole[1,5]pyrazine analog 30 was found to be a potent TYK2 enzyme inhibitor at 10 nM. An X-ray crystal structure of compound 30 bound to TYK2 confirmed the expected binding mode with polarized C7-H engaged in an interaction with the backbone C=O of TYK2 Val981. As proved by this case study, pyrazole[1,5]pyrazine scaffold could be potentially used as a kinase hinge binding motif for discovery of kinase inhibitors, especially for that requiring BBB-penetration.

Figure 3. C7-H of pyrazole[1,5]pyrazine was used as a nonconventional hydrogen bond donor. (PDB code: 6X8F)


To better understand the origin of the increased potency and subtype selectivity of CK1δ inhibitor 31, an X-ray structure of compound 31 bound to CK1δ was obtained. It was found that polarized C-H of lactam motif formed a nonconventional hydrogen bond with the backbone C=O of Leu85. Meanwhile, polarized C-H of pyridine formed an additional nonconventional hydrogen bond with the backbone C=O of Glu83 (Figure 4). [4] Scaffold 6,7-dihydropyrrolo[3,4-b]pyridine-5-one could be utilized in the discovery of inhibitors of more kinases since it potentially affords three hydrogen bonds with kinase hinge region.

Figure 4. 6,7-Dihydropyrrolo[3,4-b]pyridine-5-one scaffold formed two nonconventional hydrogen bonds with CK1δ hinge region. (PDB code: 5W4W)

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References

[1] Martin Juhas; et al. Molecular interactions of pyrazine-based compounds to proteins. J. Med. Chem. 2020, 63, 8901-8916.

[2] David A. Candito; et al. Discovery and optimization of potent, selective, and brain-penetrant 1-heteroaryl-1H-indazole LRRK2 kinase inhibitors for the treatment of Parkinson’s disease. J. Med. Chem. 2022, 65, 16801-16817.

[3] Brian S. Gerstenberger; et al. Discovery of tyrosine kinase 2 (TYK2) inhibitor (PF-06826647) for the treatment of autoimmune diseases. J. Med. Chem. 2020, 63, 13561-13577.

[4] Travis T. Wager; et al. Identification and profiling of a selective and brain penetrant radioligand for in vivo target occupancy measurement of Casein kinase (CK1) inhibitors. ACS Chem. Neurosci. 2017, 8, 1995-2004.


About Author

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By Jin Li

Senior Director of PharmaBlock


  • 10+ years’ experience in organic chemistry
  • 3+ years’ experience in medicinal chemistry
  • 10+ patents and papers published
  • Inventor of 2 clinical candidates
  • Email: [email protected]????????


Find out more at www.pharmablock.com


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